OEA sends hunger-curbing messages to the brain to increase feelings of fullness.
OEA发出饥饿抑制信息到大脑,增加饱胀感.
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OEA在骨骼肌 、 游离肝细胞 、 和主要心肌细胞的培养菌中强化脂肪酸氧化.
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When cell receptors activated by OEA were blocked, memory retention effects decreased.
当OEA激活的细胞受体被阻断后, 记忆效果就下降了.
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Under the same conditions, OEA had no effect on glucose uptake or oxidation.
在相同的条件下, OEA不 影响葡葡糖摄取或氧化.
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Currently, Piomelli said, drugs that mimic OEA are in clinical trials for triglyceride control.
目前, Piomelli说, 针对甘油三酯控制的类似OEA药物正处于临床试验阶段.
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研究人员用两种不同的检测方法发现啮齿动物食用OEA后可改善记忆.
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