1、

The protect function of MSCs to DA is considered to be relevant with the cell factor that MSCs secrete, which is the one of the mechanism of reducing neuronal apoptosis.

MSCs对DA能神经元的保护作用考虑与其能分泌的细胞因子有关,可能是MSCs减少神经元凋亡的机制之一。

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2、

Objective : To explore the effect of ginsenosides on apoptosis in cord blood cells.

目的: 探讨人参皂甙对脐血细胞凋亡作用的影响.

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3、

Objective To study the relationship between cell apoptosis and vascular disease.

目的探讨细胞凋亡与血管病变的关系.

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4、

The frequency of apoptosis in pancreatic cancer tissues was 72.6% ( 45/ 62), with degree of (+)~(+++).

在62例胰腺癌组织中细胞凋亡阳性率为72.6%(45/62),程度为(+)~(+++)。

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Effects of Carthamus tinctorius injection on bcl-2, caspase-3 expression related to neurons apoptosis after local cerebral ischemia

红花注射液对大鼠局灶性脑缺血后梗死体积和神经元凋亡相关蛋白bcl-2,caspase-3表达的影响

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7、

It'stimulates endothelial cell proliferation and vessel hyperpermeability, promotes cell migration and inhibits apoptosis.

它能促进内皮细胞增殖、迁移,阻止内皮细胞凋亡、管腔网状结构退化,增加血管渗透性.

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8、

Appearance of early cell apoptosis and reduction of Bcl-2 expression were observed at dose of 0.05mg/ mL;

在0.05mg/mL组出现细胞早期凋亡现象,Bcl-2表达明显减少;

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9、

Changes and Mechanism of Apoptosis-Related Gene Expression in T Lymphocytic Leukemia JM Cells Induced with Matrine

苦参碱诱导T淋巴细胞白血病JM细胞凋亡相关基因表达变化及其机制的研究

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10、

Flow cytometric analysis of topotecan-inhibited JM cell proliferation and topotecan-induced apoptosis

金喜素抑制JM细胞增殖并诱导其凋亡的流式细胞术分析

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Matrine Effects on JM Cells by Inhibiting Proliferation and Inducing Apoptosis

苦参碱抑制JM细胞株增殖和诱导凋亡的研究

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12、

Paclitaxel is new broad-spectrum anti-tumor drug, which has a unique mechanism of promoting the stability of microtube and inducing apoptosis.

紫杉醇是新型广谱高活性抗癌药,其独特的作用机理是促进微管聚合并稳定微管结构,诱导肿瘤细胞凋亡。

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Conclusion: A specific concentration of solanum dulcamara water extract can inhibit SPC-A-1 cell proliferation and can induce apoptosis of SPC-A-1 cells. The mechanism of action is related to expression of the bcl-2 gene.

结论:白英水提取物对人肺腺癌SPC-A-1细胞株有抑制增殖、诱导其凋亡作用,细胞凋亡的机制可能与细胞凋亡相关基因Bcl-2表达下调有关。

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15、

The expression of the apoptosis related gene bcl-2 after treatment with 50.0 mg/ ml solanum dulcamara extract for 48h ( 24.41% ± 2.96) was lower compared to untreated cells ( 53.53% ± 4.80)( P < 0.001).

50.0mg/ml白英提取物处理48h后的细胞凋亡相关基因Bcl-2表达率(24.41±2.96)%比未经处理的细胞(53.53±4.80)%表达率低(P<0.001)。

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