ischemia的例句大全

Focal cerebral ischemia was induced by middle cerebral arterial occlusion ( MCAo).

以线栓法制备左侧大脑中动脉阻塞（MCAo），诱导大鼠局灶性脑缺血模型。

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Conclusion The modified MCAO model is a cerebral ischemia-reperfusion model in rats with favorable stability and high success rate.

结论改良的大鼠MCAO模型是一种稳定性好、成功率高的脑缺血-再灌注模型。

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Methods: A rat model of focal cerebral ischemia was made by MCAO of electric coagulation.

方法：电凝阻断大脑中动脉法制备大鼠局灶性脑缺血模型。

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The rat MCAO ischemia/ reperfusion model was made by the intraluminal suture method.

采用线栓法阻断大鼠大脑中动脉产生局灶性脑缺血再灌注模型；

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Conclusion Ischemia stroke sequela model of rats can be established in 5 weeks after MCAO.

结论：大鼠MCAO5周后为缺血性中风后遗症大鼠模型。

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METHODS: The rats were randomized into 4 groups: ① The control without ischemia ( 10 rats): Vertebral artery was ligatured bilaterally and common carotid artery was not clipped bilaterally.

方法：大鼠随机分为四组：①非缺血对照组（10只）：仅结扎双侧椎动脉而不夹闭双侧颈总动脉。

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Effect on Therapy and Expression of Caspase-3 of Baicalin in Rats with Cerebral Ischemia

黄芩苷对脑缺血大鼠的治疗作用及对Caspase-3的影响

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BrdU-labelled NSCs were transplanted into the penumbra zone 3 days after cerbral ischemia. The Neurological Severity Score ( NSS) tests were undertaken in all the 3 groups 2-12 weeks after transplantation.

移植后2～12周以神经损害严重程度评分（NSS）评价各组动物神经功能状况。

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Objective: To evaluate whether supplementation of folic acid, vitamin B 6 and vitamin B 12 decreases plasma homocystine ( Hcy) level in patients with essential hypertension and ischemia stroke.

目的：探讨叶酸、维生素B6、腺苷辅酶维生素B12对原发性高血压伴缺血性脑卒中患者血浆同型半胱氨酸（Hcy）水平的影响。

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Conclusion Ani have a remarkable protection on cerebral ischemia reperfusion damage. Its mechanism may be related with decreasing the neuronal apoptosis.

结论Ani可减少脑缺血再灌注神经元凋亡，提高神经元的存活数目，对脑缺血再灌注损伤起保护作用。

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AIM: To study the protective effect of anisodine ( Ani) on acute forebrain ischemia-reperfusion injury in rats.

目的：研究樟柳碱对大鼠急性脑缺血及再灌注损伤的影响。

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The analysis of Q TC interval changes in treadmill exercise induced myocardial ischemia

平板运动试验诱发心肌缺血Q-Tc的变化分析

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Promoting effect of db-cAMP on axon regeneration and motor function recovery in brain of ischemia-reperfusion rats

双丁酸环腺苷酸促进脑缺血再灌注大鼠轴突再生和运动功能恢复

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The research of the mechanism involves in biochemistry, molecular biology and blood dyscrasia, but it's center still is high blood sugar which causes a series changes of ischemia and hypoxia.

发病机制的研究涉及生物化学、分子生物学、血液异常的等方面，但其中心环节是高血糖和其引起组织缺血缺氧发生的一系列改变。

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Change of Apoptosis Controlling Gene in Cerebral Ischemia Reperfusion Injury and Its Significance

脑缺血再灌注损伤中凋亡调控基因表达的变化及意义

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The coronary ischemia threshold reached minimum at the 10th minute.

冠脉缺血阈值与上述指标相反，10min时为最低。

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Hepatic ischemia reperfusion injury and protective effects with infusion of ATP MgCl 2 and oxygenated crystalloid solution and the observation of hepatocellular pathologic changes

肝缺血再灌注损伤和ATP-MgCl2含氧晶体灌注液的保护作用&肝细胞病理变化观察

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The frequency of ischemia attacking, paroxysmal cumulative time, motion related incidence were lower in the treated group after treatment than those in the control group.

治疗组治疗后心肌缺血发作频率、发作累计时间、与活动有关发生率均明显低于对照组；

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The positive criteria of treadmill exercise testing ( TET) mainly reflect the changes induced by myocardial ischemia in biochemical, electrophysiological and hemodynamic three aspects.

平板运动试验（TET）的阳性标准，虽有不少，但均反映心肌缺血引起的生化、电生理及心肌收缩力3个方面的变化。

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Acute incomplete cerebral ischemia was induced by ligaturing bilateral carotid arterise;

小鼠双侧颈总动脉结扎引起急性不完全脑缺血模型，测定小鼠死亡时间；

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