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CONCLUSION CVF can induce significantly exudative pleurisy sensitive to anti inflammatory agents.
结论CVF胸膜腔内注射能引起对常用的抗炎药敏感的渗出性胸膜炎。
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Under CVFF field, we optimize the full molecule by means of steepest descent and conjugate gradient.
在CVFF力场下结合最陡下降法和共轭梯度法对整个区域经过分子力学优化、分子动力学常温动态模拟获得ENOLASE烯 醇酶完整结构.
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The CVF was reduced on day 28 in treatment group when compared with AMI group ( P < 0.05).
28d时，治疗组梗死区CVF明显低于AMI组（P<0.05）；
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双向免疫电泳鉴定,发现CVF与 人血清作用后,其中补体成分C_ 3分子的抗原性发生改变,则表明CVF的作用是通过激活补体成分C_3而发挥的.
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There was no relationship between left atrial CVF and left ventricle diastolic pressure.
左房CVF与左室舒张压之间无直线相关关系（r＝0.1691，P＞0.05）。
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Thus, it is clear that afford ability is seen as the critical issue for CVF.
因此, 很明显CVF的资金保障能力看起来是有限的.
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