1、

Objective: To analyze the interactions between AChR γ subunit gene and nuclear factors present in developing chicken skeletal muscle.

目的:分析鸡乙酰胆碱受体(acetylcholinereceptors,AChR)γ亚基基因与不同发育阶段鸡骨骼肌核因子的相互作用。

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2、

Specific T cell mediated immunity against AChR in peripheral blood monocytes in patients with myasthenia gravis expression of sIL 2R, IFN γ and IL 4 and transcription of their mRNA

重症肌无力患者外周血乙酰胆碱受体特异性T细胞sIL-2R、IFN-γ、IL-4的表达和转录

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3、

Construction and Screening of TsF cDNA Library Specific for AchR

AchR特异性TsF cDNA文库的构建及筛选

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4、

AChR subunit mRNA expression in human muscles of patients with myasthenia gravis

重症肌无力患者肌肉AChR亚单位基因的表达

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5、

And could the activated TDCs present antigen AChR especially in MG thymus?

活化TDC能否提呈MG特异性抗原-胸腺组织中AChR蛋白?

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6、

Difference of T cell immune response to AChR in Lewis rats and Wistar Furth rats

Lewis和Wistar Furth大鼠T细胞对AChR的免疫应答不同

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7、

The downregulation of specific T Lymphocyte responses by nasal administration of AChR in EAMG rats

鼻腔AChR耐受低调EAMG大鼠特异性T淋巴细胞反应

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8、

CONCLUSION The human anti-AChR antibodies, secreted from the mice transgenic for human Ig loci, induced with Torpedo AChR, are able to bind to AChR in the mouse muscles, which leads to the damages of AChR, resulting in the animal EAMG.

[结论]电鳐乙酰胆碱受体刺激人免疫球蛋白转基因小鼠产生的人抗乙酰胆碱受体抗体,可与肌肉组织中的乙酰胆碱受体结合,导致肌肉收缩无力,可诱导重症肌无力。

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