Conclusion Bcl-2 gene family may participate in the DATS induced apoptosis of MGC-803 cells.
结论Bcl -2 基因家族可能参与了DATS诱导的MGC -803 细胞凋亡.
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结论: 那仁满都拉-11,有抑制肿瘤的作用, 可能与bcl -2 蛋白上调有关.
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The expression of p 185 was reversely correlated to that of bcl-2 in ChC.
p185表达与bcl -2 表达呈负相关.
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The staining of TOPO-II α, P-gp, GST-π and BCL-2 are no significant different between them.
而TOPO- 、 P-gp 、 GST - π、BCL -2 的表达差异无显著性.
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Conclusion: QY and YP can lower the expression of Bcl-2 but not act on P 53.
结论: 清毒饮、养正片确能下调Bcl -2 表达,而对P53作用不够明显.
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The expression of Fas increased with PYM concentration while the expression of Bcl-2 decreased.
Fas表达随药物浓度增加而递减,Bcl -2 表达随药物浓度增加而升高.
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The Study on Effect of Apoptosis-related Gene C-myc, bcl-2 and p 53 in Infancy Hemangiomas.
凋亡相关基因C-myc, bcl -2 ,p53在小儿血管瘤中的作用研究.
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The expressions of bcl-x, bax and eNOS were measured by SQ-RT-PCR technique.
采用半定量逆转录聚合酶链反应(SQ-RT-PCR)法检测内皮细胞bcl-x 、 bax以及 eNOS表达.
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So to a certain extent, bcl-2 and bax expression might reflect the DG and biological behavior.
推测bcl -2 、bax表达水平在一定程度上反映了肿瘤的生物学行为,可作为预测预后的指标.
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Conclusion: Bcl-2 was restraining apoptosis gene, Bax, Bak were promoting apoptosis gene.
结论: Bcl一2为抑凋亡基因, Bax和 Bak为促凋一亡基因, 它们可以作为评估喉鳞癌生物学特性的参考指标.
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Conlusion 1 Over expression of Bcl-2 was observed in biliary carcinomas.
结论1Bcl -2 蛋白在胆管癌与良性胆管疾病中表达有显著差异,它的过度表达与肝外胆管癌的发生有关.
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The lev-els of Bcl-2 and Bcl-X _ L proteins were examined by western blot analysis.
Westernblot法分析细胞内Bcl -2 和Bcl -X _L蛋白水平的变化.
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结论Bcl -2 、 Bcl-xl 、 Bax蛋白可能参与了基底细胞癌的发生、发展.
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Results YZ decreased cytochrome C expression and ICE mRNA content and increased Bcl-2 mRNA content.
结果益智口服液可以降低细胞色素C蛋白质表达、减少白介素-1β转化酶mRNA含量、升高B细胞淋巴瘤mRNA含量。
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bcl-2的表达随病理分级升高和临床分期进展而降低,而与淋巴结转移无关(P>0.05);
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9/ 32 ( 28.1%) nodal DLBCL had the bcl-6 gene rearrangement, but none of RLH did.
9/32(28.1%)DLBCL有bcl-6基因重排,而RLH中均未检测到bcl-6基因重排;
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结果表明,饱和脂肪酸通过其代谢产物酰基辅酶A呈剂量依赖性的引起颗粒细胞凋亡,并导致Bcl2水平下降和Bax水平升高。
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目的:观察新风胶囊对佐剂性关节炎大鼠疗效及对病变滑膜fas、fasL、bcl2表达的影响。
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结果:BAX mRNA在流产胎盘中的表达量明显高于对照组胎盘。在胎盘绒毛滋养层细胞可见明显表达,基底层蜕膜细胞也有表达。
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