1、

Conclusions Tetrandrine could promote apoptosis of neuroblastoma cell line TGW and up-regulate the expression of Bax gene.

结论汉防己甲素能诱导人神经母细胞瘤株TGW凋亡,上调Bax基因的表达。

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2、

These studies have shown that: TRX can suppress tumor cell apoptosis.

研究表明:TRX可以抑制肿瘤细胞凋亡。

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3、

Trx plays crucial roles in regulating cell growth, apoptosis and gene transcription.

Trx具有调节细胞生长、抑制凋亡、调节基因转录等功能。

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4、

In this paper we discussed the mechanism of apoptosis induced by TRG in tobacco cells.

探讨了TRG诱导烟草细胞凋亡的机制。

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5、

Programmed cell death ( PCD) in tobacco cells induced by TRG showed the typical features of apoptosis.

TRG诱导的烟草细胞死亡表现为细胞凋亡的典型特征。

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6、

The effect of TFu on the apoptosis and proliferation of FRH-0201 cholangiocarcinoma cell line

TFu对肝门部胆管癌细胞系FRH-0201增殖和凋亡的影响

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7、

Conclusion The combined use of RSG and CDDP can induce apoptosis of A549 cells and it shows evidently time and dose dependence.

结论罗格列酮与顺铂合用能诱导肺癌A549细胞调亡,呈明显的时间及剂量依赖性。

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8、

Conclusion Early observation of △ψ m in RGC apoptosis has certain pragmatic value, especially on quantitative analysis.

结论△ψm在RGC凋亡早期的观测中有一定的意义,并且能进行定量分析研究。

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9、

PMX caused DNA damages in murine embryonic stem cells, oxidative stress injuries of mice and apoptosis of embryonic stem cells, leading to abnormal closure of neural tube and ultimately NTDs.

体外实验中,PMX可以损伤小鼠胚胎干细胞DNA,造成小鼠氧化应激损伤,使胚胎干细胞发生凋亡,最终使神经管不能发生正常闭合,而导致神经管畸形的发生。

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10、

Targeted activation of protein kinase PKR induced apoptosis of leukemia K562 cells

靶向激活蛋白激酶PKR诱导白血病K562细胞凋亡

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Expression of PKB and apoptosis in pancreas of diabetic rats

糖尿病大鼠胰腺中蛋白激酶B表达和细胞凋亡的变化

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12、

OFQ can induce the apoptosis of K562 cells. 3.

孤啡肽可诱导K562细胞凋亡。

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13、

The study on the expression of Msx gene 、 BMP-4 and apoptosis during mouse tooth morphogenesis

小鼠牙齿形态发生过程中Msx1/2、BMP-4的表达及细胞凋亡的研究

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14、

Effect of cadmium on early apoptosis of NRK cells and gene expression of bax

镉诱导大鼠肾细胞早期凋亡及bax基因表达的实验研究

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Effect of Cadmium Chloride on Apoptosis Morphology of NRK Cells

镉对正常大鼠肾成纤维细胞凋亡形态学的影响

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Apoptosis rate of NRK cell treated by serum of different groups were assessed by flow cytometry methods.

体外培养大鼠正常肾NRK细胞,用上述4组SAP大鼠血清分别置换培养液中小牛血清,流式细胞术检测处理后细胞凋亡率。

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17、

Effects of MAPK inhibitors on mRNA expression of apoptosis genes in NRK cells treated with cadmium chloride

p38MAPK和ERK1/2抑制剂对镉诱导肾细胞凋亡基因表达的影响

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18、

Conclusion CdCl_2 could induce early apoptosis of NRK cells and up-regulate the expression of bax mRNA.

结论CdCl2可以诱导NRK细胞早期凋亡,并可使bax基因表达增强。

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19、

Conclusion Stimulation with LPU could induce cell proliferation at a shorter time and could inhibit cell growth or even cause cell apoptosis in vitro at a longer time.

结论短时间低剂量脉冲超声波刺激能促进细胞增殖,超过一定时间则会抑制细胞生长,甚至引起细胞凋亡。

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20、

Conclusion: The LLN can alleviate rat myocardium from injury by increasing bcl-2/ bax, decreasing the expression of caspase-3 and reducing myocardial apoptosis.

结论:LLN对异丙肾上腺素造成的大鼠急性心肌损伤有保护作用,其机理与升高bcl2/bax比值,降低caspase3的表达和抑制凋亡有关。

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