1、

Immunohistochemical ABC method was applied to display the GABA and NF immunoreactive ( GABA-IR and NF-IR ) structure.

运用免疫组织化学ABC法显示γ-氨基丁酸 ( γ-Aminobutyricacid,GABA ) 、神经丝蛋白( neurofilament,NF)阳性结构.

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2、

A planar film of refractive index nf is sandwiched between a substrate and a cover material.

折射率为nf的平面薄膜夹在折射率较低的衬底和覆盖层之间.

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3、

How XIAP inhibits caspases is known, but how XIAP induces NF-kappa B actiation is entirely unclear.

XIAP怎样抑制Caspases是已知的, 但是XIAP怎样诱导NF - κB活化是未知的.

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4、

NUE of three types of NF exist genotype difference.

各类型氮肥的利用率均具有基因型差异.

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5、

CONCLUSION: NF-κ B decoy ODNs will become a new gene drug for treating inflammatory bowel disease ( IBD ) .

结论: NF - κ BdecoyODNs 有望成为治疗IBD的一种新型基因药品.

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6、

TLR 4 induces activation of transcription factor NF-κ B and regulates transcription of a variety of cellular genes.

TLR4通过活化下游分子核因子-κB( NF-κB)调节靶基因转录.

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7、

NF Series are dedicated controller series which the standard function blocks are used.

NF系列是使用标准功能块的专用控制器系列.

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8、

Furthermore, CpG methylation within the NF 2 promoter region was thoroughly analyzed.

此外, 充分分析了NF2启动子区域内的CpG甲基化.

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9、

Conclusions NF-κ B may minimally affect the progression and invasion of COC.

结论NF - κB在COC的恶性转化及侵袭过程中影响较小.

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10、

NF-κ B inhibitor CAPE suppressed LPS-induced these effects.

NFκB抑制剂咖啡酸苯乙酯(CAPE)抑制了LPS介导的上述效应.

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11、

Results; NF protein in the hippocampus only labeled some neurons.

结果: 在海马结构内并不是所有的神经元均可被神经丝蛋白所标记.

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12、

ASI can lower expression of NF-kB and decrease cerebral ischemia-reperfusion injury.

刺五加注射液可下调的NF-kB表达,减轻脑缺血再灌注损伤.

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13、

Immunohistochemistry and Western blotting were used to detect the expression of neurofilament ( NF ) and neuron specific enolase ( NSE ), respectively.

免疫组织化学染色法和Westernblotting法检测神经丝蛋白 ( NF ) 和神经元烯醇化酶 ( NSE ) 的表达.

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14、

Objectives : To observe the roles of nuclear factor κ B ( NF κ B ) in corticosterone injured hippocampal neurons.

目的: 探讨核因子κB ( NFκB ) 在皮质酮损伤海马神经元中的作用.

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15、

Objective To investigate the role of NF KB , NCAM in epileptogenesis of Pentylentetrazol kindling rats.

目的 探讨核因子κB( NFκB) 、 神经细胞粘附分子(NCAM)在戊四氮(PTZ) 点燃大鼠点燃前即癫痫形成过程中的作用.

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16、

Conclusion: PC-1 NF fusion protein may inhibit the proliferation, induce apoptosis and decrease ECM production of RMC.

结论PC-1NF融合蛋白能抑制大鼠系膜细胞增殖 、 诱导细胞凋亡及减少细胞外基质合成.

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17、

Including Bond Wire and Pad effects both, detailed performances have also been proposed with NF deterioration to 3.21/3.23 dB, but S parameters change a little after tuning and optimization, finally, the circuit turns out to have outstanding performances.

完全考虑Bond Wire和Pad效应的性能参数也已经给出,但噪声系数恶化为3.21/3.23dB,S参数在电路调整优化之后变化不大,整体性能比较突出。

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18、

Conclusion The increased NF-κ B expression has both protective and harmful effects on neuronal cells after global ischemia-reperfusion in rats.

结论NF-κB在大鼠全脑缺血再灌注后神经细胞中的表达增加,可能具有保护和损伤双重作用。

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19、

Effects on Cell Proliferation and NF-κ B Pathway in Neuroblastoma after Inhibition of Notch Pathway by DAPT

DAPT抑制Notch信号通路对神经母细胞瘤细胞增殖的影响

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20、

The damages of membrane proteins and DNA, mitochondrial function, activation of NF κ B and expression of IL 6 were studied.

分析活性氧对细胞膜蛋白和DNA的损伤,细胞线粒体功能变化,白介素6(IL6)的表达变化以及细胞核因子κB(NFκB)的激活。

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