1、

The image analysing result: The TH+ neuron numbers in VTA and SNC at five level brain sections were significantly different among three groups ( P < 0.05).

免疫组化定量分析:在5种不同水平的中脑切片中,三组大鼠中脑VTA和SNC TH十神经元数量均有显著性差异(P<0.05)。

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2、

These specialised nerve cells mimic the mental state of those we come in contact with.

这些特定的神经元细胞可以模拟我们所接触到的其他人的心理状态.

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3、

The diagram shows the electrical connections that each of the 302 neurons makes to others in the system.

图表显示系统中302个神经元每个都和其他的形成电连接。

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4、

The Changes of Plasticity of Microglia and Astroglia in the Degeneration and Protection of Dopaminergic Neurons

小胶质细胞和星形胶质细胞在多巴胺能神经元变性和保护中的可塑性变化

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5、

CONCLUSION Free radical produced by self-oxidation of bendopa can promote development of Parkinson disease. APS can relieve the toxicity of bendopa on neurons and the effect has time dependency.

结论:左旋多巴自身氧化产生大量的自由基,促进帕金森病发展,黄芪多糖可降低左旋多巴对神经元的毒性作用,作用有时间依赖性。

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6、

Excitative effect of TNF α-activated astrocytic conditioned medium on hippocampal neurons

TNFα刺激的星形胶质细胞条件培养液对海马神经元的兴奋作用

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7、

Protective effects of caffeine on apoptosis of cerebellar granule neurons induced by diphenylhydantoin

咖啡因对苯妥英诱导的小脑颗粒神经元凋亡的保护作用及机制

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8、

The results suggested that the increased endothelin level of ischemia brain territory was important factor leading to ischemic neuronal injury, and FNZ had protective effects on cerebral ischemia.

上述结果提示:缺血脑组织内皮素含量升高是导致缺血神经元损伤的重要因素,氟桂嗪对脑缺血有保护作用。

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9、

Objective To study the dual effects of calcium antagonists on ischemic hypoxic injury of hippocampal neurons cultured in vitro by using flunarizine ( FNZ) and the mechanism of its action.

目的利用氟桂利嗪(FNZ)研究钙离子拮抗剂对体外培养海马神经元缺血缺氧损伤的作用及其作用机制。

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10、

The steady state probability distribution and mean first passage time of FHN neural system driven by non-Gaussian noise

非高斯噪声激励下FHN神经元系统的定态概率密度与平均首次穿越时间

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11、

Second, we study the effect of noise in the information code of HH and FHN neuronal systems.

其次,我们以FHN和HH模型为例研究了在神经元信号编码过程中噪声的影响。

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12、

Conclusion YC 1 can block apoptosis of cerebellar granule neurons induced by low K+. The neuronal protection of YC 1 maybe belongs to a nongenomic action.

【结论】YC1抑制低钾诱导小脑颗粒神经元凋亡,YC1的这一作用可能属于一非基因效应。

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13、

Results showed that the neuronal survival was 199.11 ± 24.00 in the high K+ group, 47.56 ± 11.09 in the low K+ group and 201.11 ± 27.43 in the FK group.

结果表明:①幸存神经元数:高钾组199.11±24.00,低钾组47.56±11.09,FK组201.11±27.43;

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14、

The relationship between the prevention of neuronal apoptosis by high K~+ and cAMP

高K~+阻止神经元凋亡与cAMP关系的研究

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15、

Aim: To investigate the roles of serotonergic neurons in dorsal raphe nuclei ( DRN) in sleep.

目的:研究中缝背核(DRN)5羟色胺(5HT)能神经元在睡眠中的调节作用。

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17、

It is showed that high glucose causes DRG neurons apoptosis.

实验证明,高糖可引起体外培养的DRG神经元凋亡。

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18、

Detect hippocampal neurogenesis with Brdu and DCX double marker. 4.

利用Brdu和DCX双重标记,检测各组海马内神经元的再生情况。

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19、

Apoptosis was induced by low potassium in cultured rat CGN.

【方法】以低钾诱导的大鼠小脑颗粒神经元凋亡为模型。

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20、

AIM: To explore the effects of carbenoxolone ( CBX), a gap junction blocker, on the neuron injury induced by focal cerebral infarct.

目的:探讨缝隙连接阻断剂甘珀酸对大鼠光化学局灶性脑梗死神经元损害的影响。

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