1、

G1 arrest and apoptosis of mouse tumor after radiotherapy and electrochemical therapy

放疗结合电化疗对小鼠肿瘤组织细胞G1期阻断与凋亡的诱导

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2、

Ectopic Expression of Cyclin G2 Inhibits Cell Proliferation in HeLa Cancer Cell Line

Cyclin G2抑制肿瘤细胞增殖的研究

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3、

Methods ( 1) I5 cases of CSTP were studied using histologic ( HE), immunohistochemical ( PV6000) methods.

方法①应用组织学(HE法)和免疫组织化学(PV6000法)对15例胰腺囊性-实性肿瘤组织进行观察及检测。

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4、

Conclusion The plasma concentration of EBV DNA could reflect the tumor burden in patients with NPC.

血浆EBVDNA 水平与TV、 NV、 TNV呈正相关关系.结论鼻咽癌患者治疗前血浆EBVDNA 水平可在一定程度上反映其体内肿瘤负荷.

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5、

Study of P3 wave in frontal and temporal lobe tumor

额叶、颞叶肿瘤事件相关电位P3的研究

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6、

So we studied the mechanism of S1 induced apoptosis in MCF-7 cells.

因此,以化合物S1作为抗癌药物先导物探讨诱导MCF-7肿瘤细胞凋亡的作用机制。

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7、

Studies on antitumor activity of A7 extracted from one fungal mycelium

一种真菌菌丝体甲醇提取物&A7的抗肿瘤活性研究

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8、

Results: The tumor volume and cell load were all differential significantly in each gade of WHO response. The relation between the WHO criteria and the LCK has been defined.

结果:每级别WHO疗效在相同或不同大小肿瘤都存在体积、细胞负荷和LCK的显著差异,WHO疗效评价与LCK相互换算关系得到确定。

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9、

Conclusion: WHO response criteria can not determine the tumor volume, the cell load as well as the LCK. A new method evaluating LCK quantitatively has been proposed and it may be interconverted with WHO response criteria.

结论:WHO标准不能定量评价肿瘤体积、细胞对数和LCK,提出可与WHO疗效评价相互换算的LCK定量评价方法非常必要。

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10、

The objective of this study was to investigate the correlation of WHO criteria with the tumor volume, the tumor cell load and the LCK; and the necessity developing a method to measure LCK quantitatively.

本研究目的是探讨WHO疗效标准与肿瘤体积或细胞负荷及细胞对数杀灭(logcellkill,LCK或LK)相互关系及提出细胞对数杀灭定量评价方法的必要性。

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11、

These results further sulgest that CSB gene is a potential target for cancertheray.

本研究为深入研究CSB基因功能和开发其为抗肿瘤靶基因奠定了基础.

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12、

The purified CTX-d without PLA 2 showed potent antitumor activity both in vivo and in vitro.

结果分离纯化获得的CTX-d 不混有PLA2,在体内外实验中显示明显的抗肿瘤作用.

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13、

DSA examination in 3 cases presented obvious staining of tumor.

行DSA检查3例,均显示明显的肿瘤染色.

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14、

DVH, DSH, TVR and tumor volume.

DVH, DSH, TVR,适形性及肿瘤控制情况.

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15、

Tumor stage was assigned by DRE results , according to the scientists.

肿瘤分期视DRE结果而评定.

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16、

The positive staining of VEGF in tumor cells and factor in new ECs was seen.

肿瘤细胞VEGF强阳性着色,新形成的血管内皮细胞因子抗体阳性.

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17、

Skp 2 is an Fbp, which targets many ubiquitin-mediated degradation of G 1-and S-phase regulators.

研究表明许多恶性肿瘤中存在着Skp2相关机制的异常, Skp2现已被认为是一种重要的癌基因.

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18、

Conclusion The uptake of 18 F-FLT in pulmonary malignant tissues is higher than that in normal tissues.

结论肺部恶性肿瘤组织中 18F-FLT 摄取高于正常组织.

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19、

Conclusions: galectin-3 and VEGF-C can be good molecular markers in FNA diagnosis of thyroid lesions.

结论: Galectin -3 和VEGF -C 检测可以为甲状腺肿瘤细胞学诊断提供有价值的分子生物学依据.

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20、

Progress of Mechanism Study on Malignant Tumor Treated by Principle of Yiqi Jianpi

益气健脾法治疗恶性肿瘤机理研究的进展

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