CDK

Cyclin-cdk compound is composed of Cyclin and cdk different cell cycle.

多种肿瘤存在着Cyclin的异常表达,cdk的激活及其抑制因子的丢失.

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Meanwhile, the activity of CDK 5 was 3.5 times higher than that in non-transfection group.

与此同时, 细胞周期蛋白依赖性蛋白激酶-5酶活性较未转染组提高3.5倍.

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Rapamycin arrested cell cycle in G 1 phase to antitumor by degrading the CDK-cyclin complexes kinase activity.

Rapamycin还能通过降低CDK-cyclin复合物激酶的活性,使细胞周期阻滞在G1期,从而发挥抗肿瘤的作用.

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Cyclin D 1 and CDK 4 proteins were expressed correctly in insect cells and had their biological activity.

成功构建了细胞周期蛋白D1及CDK4真核杆状病毒表达载体,并且在昆虫细胞中正确表达了具有生物活性的细胞周期蛋白D1及CDK4融合蛋白.

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CDK activation by cyclin binding.

1)CDKs结合cyclin后活化.

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Protein serine theronine phosphatase inhibitor or cyclin dependent kinase ( CDK ) inhibitors could abolish the enhancement.

而蛋白丝氨酸苏氨酸磷酸酯酶抑制剂以及细胞周期蛋白依赖性激酶 ( dk ) 制剂可阻断该凋亡敏感性增强效应.

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Cell cycle is mainly regulated by cyclin, cyclin dependent kinase ( CDK ) and cyclin dependentkinase inhibitor ( CDKI ).

细胞周期主要是由细胞周期蛋白 ( cyclin), 细胞周期蛋白依赖性激酶 ( CDKs ) 和CDK抑制蛋白(CDKI ) 来调控的.

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Objective To construct recombinant expressing Cyclin D 1 and CDK 4 proteins.

[目的]建表达人细胞周期蛋白D1及细胞周期蛋白激酶CDK4融合蛋白的重组体.

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Cell cycle in plants is controlled byactivity of cyclin-dependent kinase ( CDK ) complexes.

植物细胞周期主要由细胞周期蛋白依赖性激酶 ( CDK ) 复合物调控.

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Objective To investigate the role of FHIT, cyclinD 1, CDK 4 in NSCLC.

目的研究非小细胞肺癌(NSCLC) 中脆弱性组氨酸三联体(FHIT) 、 细胞周期素D1(cyclinD1) 、 细胞周期蛋白依赖激酶4(CDK4)的表达与NSCLC发生发展的关系.

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CDK and cyclin are key molecules that control and coordinate DNA-synthesis, chromosome separation and cell division.

CDK和细胞周期蛋白是控制和协调DNA合成 、 染色体分离和细胞分裂的重要分子.

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The Role of CDK 5 in the Pathogenesis of OPIDN.

CDK5在有机磷致迟发性神经病中的作用.

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Therefore, DOC-1 R gene may be an CDK 2 inhibitor specifically.

因此DOC -1R 可能为CDK2专一性的CDK抑制 因子(CDKinhibitor,CK1).

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In the YIQIJIEDU granule group, some genes in array were lowly expressed , such as STK 1, CDK , MAPK, et al.

HumanCancercDNAArray分析发现,益气解毒颗粒组有STK1 、 MAPK1 、 CDK1等19个与细胞增殖、细胞周期 、 核转录因子、DNA修复 相关基因表达下调.

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