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采用免疫组织化学和生化法检测心肌ICAM -1 的表达及髓过氧化物酶 ( MPO ) 水平.
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MoAb to CD 11 b and ICAM-1 significantly inhibited this adhesiveness ( P < ( 0.05 )).
CD11b单抗和 ICAM1 单抗可明显抑制这种粘附 ( P<0.05 ).
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提示 抗 ICAM -1 单抗对异丙肾引起的心肌损害具有保护作用.
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Results showed that ICAM-1 immunoreaction was negative or weak in normal control group.
结果显示正常对照组大鼠脑血管ICAM -1 免疫组织化学显色为阴性或弱阳性反应.
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目的探讨创伤患者急性期C肽 ( CP ) 、 细胞间黏附分子 -1 ( ICAM -1 ) 、 胰岛素抵抗(IR)的变化特点及其临床意义.
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The expression of ICAM-1 mRNA can be inhibited when treated with GCV and PFA ( P < 0.05 ).
GCV和PFA可 抑制HCMV感染诱导的ICAM -1 表达的上调 ( P<0.05 ).
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ICAM-1 may play important roles in myocardial ischemia-reperfusion injury after CPB.
ICAM -1 在CPB后心肌缺血再灌注损伤中起重要作用.
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Results: 1 . The protein and mRNA of ICAM-1 was expressed at low levels on nomoral TEC.
结果: 1. 正常大鼠肾小管上皮细胞ICAM -1 蛋白质和mRNA低水平表达.
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Continuous ICAM 1 expression might be related to the stenosis of eustachian tube.
咽鼓管狭窄可能是粘膜ICAM- 1持续表达的原因之一.
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Asarinin could also decrease the level of ICAM-1 and VCAM-1 in the allografts.
CsA组 、细辛脂素组和混合药物组移植心局部ICAM -1 和VCAM -1 含量较阳性对照组明显减少.
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