1、

CONCLUSION SAB and Curcumin could significantly inhibit the proliferation, activation and ERK phosphorylation.

结论SAB和Cur能够抑制HSC的增殖 、 活化和ERK的磷酸化.

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2、

Treatment with CHEL significantly increased hCG accumulation in JAR cells , and enhanced the phosphorylation of MAPK.

对照组及各实验组都有活性的MAPK的 表达,与对照组相比,PMA慢性 刺激组、CHEL组MAPK磷酸 化水平升高;PMA急性 刺激组MAPK磷酸化水平降低.

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3、

Alterations in the content, distribution and phosphorylation status of gap junctions are correlated with ventricular arrhythmia.

缝隙连接的含量 、 分布及自身磷酸化状态的改变与室性心律失常的发生密切相关.

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4、

Objective : To investigate the phosphorylation of KCNE 2 protein in heart of old SHR rats.

目的: 探究在高龄SHR大鼠 心室膜蛋白中,KCNE2蛋白是否发生了磷酸化修饰.

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5、

Overexpression of c-Src leads to tyrosine phosphorylation and cell transformation of many kinds of protein substrates.

过表达 Src 可以导致多种蛋白质底物的酪氨酸磷酸化以及细胞转化.

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6、

The reversible phosphorylation of proteins is important in intercellular communication.

蛋白质的可逆磷酸化作用在细胞间的信息传输中非常重要.

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7、

Deltamethrin was found to affect ATPase activity and protein phosphorylation of housefly brain synaptosome in vitro.

为材料,研究和比较了神经毒剂溴氰菊酯对其脑突触体蛋白磷酸化作用的影响.

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8、

The tyrosine phosphorylation of leptin receptor was revealed by immuno precipitation.

应用免疫沉淀法检测瘦素受体的酪氨酸磷酸化程度.

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9、

Genistein, the tyrosine kinase inhibitors, plays the effect by an inhibition of tyrosine phosphorylation.

Genistein 是酪氨酸激酶的非特异性抑制剂, 通过抑制酪氨酸的磷酸化而发挥作用.

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10、

Leigh syndrome is a severe early-onset progressive neurodegenerative disorder due to mitochondrial oxidative phosphorylation defects.

Leigh综合征是一种由于线粒体氧化磷酸化障碍所导致的严重退行性脑病.

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11、

Thirteen of these genes provide instructions for making enzymes involved in oxidative phosphorylation.

十三这些基因提供指令,为决策所涉及的酶的氧化磷酸化.

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12、

MAPK inhibitor ( PD 98059 ) inhibited the phosphorylation of GSK-3 P and almost completely blocked oxLDL-induced tubular EMT.

MAPK抑制剂 PD98059 可抑制GSK -3 β的磷酸活化,并几乎完全阻断oxLDL诱导的肾小管上皮细胞转分化,但对LDL诱导 的小管上皮细胞转分化只有部分阻断作用.

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13、

Aim To study effects of cell protein phosphorylation on scavenger receptor.

目的观察细胞内蛋白磷酸化水平对清道夫受体功能的影响。

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14、

The function of the cardiac GJ is regulated by different factors including intracellular PH, concentration of intracellular Ca~ ( 2+) and ATP, the phosphorylation status of the Cxs, the voltage difference between two end of the channel, some neurohumoral factors and etc.

心脏缝隙连接通道的功能受胞内pH值、胞内Ca2+浓度、ATP浓度、连接蛋白的磷酸化状态、跨通道电压和一些神经体液因子等的调控。

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15、

The level of phosphorylation of Erkl/ 2and Akt signaling molecules further increased by HNK may also be one of the mechanisms.

而HNK使得Erkl/2及Akt信号分子的磷酸化水平进一步增加可能也是其作用机制之一。

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16、

The tyrosine-specific protein phosphatase ( Yersinia enterocolitica protein, YOP) could significantly inactivate the phosphorylation activity of protein kinase.

酪氨酸特异性蛋白磷酸酶(YOP)对该激酶的活性有显著的钝化作用。

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17、

The Alternation of Tyrosine Protein Phosphorylation of HL-60 during its Denucleation Induced by EDDF

红细胞分化因子诱导HL-60细胞排核过程中酪氨酸蛋白质磷酸化的改变

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18、

The main mechanism of apoptosis induced by object gene protein overexpression of PDK molecule's Pleckstrin homology domain is to reduce PKB molecule's phosphorylation level of apoptosis-resisting cells, thus decrease their activity and unable to play an active biochemical role.

PDK分子PH结构域目的蛋白的过表达引起的细胞凋亡的机制主要是下调了起抵抗细胞凋亡作用的PKB分子的磷酸化水平,从而降低其活性,无法发挥积极的生化作用。

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19、

Consequently, we hypothesized that the relative lack of adhesion matrix may be contributed to the decrease of PKB phosphorylation and the following S-phase arrest in the β 1-7721 or α 5 β 1-7721 cells.

因此,对α5β1-7721和β1-7721细胞而言,粘附基质的相对不足,可能就是PKB磷酸化水平降低和细胞周期抑制的原因之一。

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20、

Conclusion IL-12, IL-2 and LPS may promote lck gene express in renal tubular epithelial cells and only IL-12 can induce p38 phosphorylation through lck uniquely, and they can play biologic role in renal tubular epithelial cells via lck/ p38 signaling pathways.

结论IL-12、IL-2、LPS促进肾小管上皮细胞中lck基因表达,并只有IL-12唯一通过lck诱导p38磷酸化,它们皆可通过lck/p38信号传递途径参与对肾小管上皮细胞发挥生物学作用。

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