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Methods: Activity of PKC was determined by using competitive protein binding method.
方法: 应用竞争蛋白结合法检测PKC活性.
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To evaluate the possible protective effects on HUVECs mechanisms of DIPA, PIO and AM involved.
观察DIPA, PIO及 AM对EC蛋白 激酶C( PKC) 亚型PKCα 、PKCδ的表达的影响,了解其可能的作用机理.
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与细胞增殖 、 分化 、 凋亡 、 心肌肥大、心室重塑等有着密切联系.
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Protein kinase C plays an important role in regulating the messages transmition acrossing membrane.
蛋白激酶C(PKC)在跨膜信息传递中起着重要的调控作用.
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TPA is a potential tumor promotor and an activator of protein kinase C ( PKC ).
佛波酯(TPA ) 是潜在促肿瘤剂,也是蛋白激酶PKC激活剂.
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There was no significant difference between IP C and IPTC groups.
IPTC组的IS、 心肌细胞膜PKCα 的表达与IPC组无显著差异(P>0.05).
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Activates protein kinase C ( PKC ) , presumably by binding to the pseudo substrate sequence.
激活PKC, 可能的机理是结合相似底物序列.
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