reperfusion

The IPC 、 PMA protected the myocardium from sustained ischemia-reperfusion injury . Bre.

Bre. 则可消除IPC对心肌的保护作用.

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Conclusion: F 2 can antagonize the arrhythmias induced by ischemia-reperfusion in rat heart.

结论: F2对大鼠心脏缺血再灌注心律失常具有拮抗作用.

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Objective To study the level of nitric NOS in ischemia-reperfusion rat liver.

目的探讨大鼠缺血再灌注肝一氧化氮合酶的变化及其意义.

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ASI can lower expression of NF-kB and decrease cerebral ischemia-reperfusion injury.

刺五加注射液可下调的NF-kB表达,减轻脑缺血再灌注损伤.

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Assessing the Myocardium After Attempted Reperfusion: Should We Bother?

试图再灌注后评估心肌: 我们应否打扰?

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ICAM-1 may play important roles in myocardial ischemia-reperfusion injury after CPB.

ICAM -1 在CPB后心肌缺血再灌注损伤中起重要作用.

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In reperfusion group, the thrombus was thinner and unclear.

溶栓组可见血栓模糊不清, 形态较血栓组稀薄.

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In the later reperfusion period, less H 2 O 2 generated. Intra-cellular calcium increased continuously.

再灌流晚期H2O2产生有所减少,而细胞内钙持续增高.

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Methods: The animals achieved hypercapnic acidosis with mechanical hypoventilation before ischemia-reperfusion.

方法在缺血再灌注前,用低潮气量,低频率的机械通气方法造成动物的呼吸性酸中毒.

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Objective : To identify the relationship of hypovolemic shock and reperfusion injury.

目的: 确定创伤所致的低血容量性休克病人预后与再灌注损伤的关系.

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Methods A model of retinal ischemia reperfusion in rat was made.

方法制备大鼠视网膜缺血再灌注模型.

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Apoptosis is an important pathophysiological characteristic during hepatic ischemia-reperfusion injury ( HIRI ) .

细胞凋亡是肝缺血 — 再 灌注 损伤重要的病理生理特征.

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Thrombolysis therapy and percutaneous transluminal coronary angioplasty recover myocardial reperfusion efficiently.

溶栓治疗和经皮冠状动脉腔内成形术有效恢复缺血心肌的再灌注.

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The destruction of BBB is an important mechanism of cerebral ischemia-reperfusion injury.

血脑屏障破坏是脑缺血再灌注(ischemiareperfusion,IR) 脑损伤的重要的病理生理基础.

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Unchecked, oxygen radicals may cause reperfusion injuries and other problems.

看来, 氧基能导致多次灌注的伤害和其他问题.

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