1、

Conclusion Melatonin can protect small intestinal mucosa barrier through decreasing the shift of intestinal bacteria endotoxin, lowering cell factor level in plasma and inhibiting ICAM-1 expression in intestinal tissue after obstructive jaundice.

结论褪黑素通过减少肠道细菌内毒素的移位,降低血浆中细胞因子水平,抑制梗阻性黄疸后肠组织ICAM-1的表达,保护小肠黏膜屏障。

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2、

Study of Injury and ICAM-1 Expression in the Rats Small Airway Epithelium

大鼠小气道损伤及细胞黏附分子表达的实验研究

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3、

Conclusion High or low expression of adhesion molecules ICAM-1 、 CD_ 44 V6 、 CD_ 28 、 E-cad and CEA plays a certain role in organic selection of gastric carcinomatous metastasis.

结论粘附分子ICAM-1、CD44V6、CD28、E-cad、CEA表达的增强或减弱在胃癌转移器官选择性中起到一定的作用。

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4、

After TCR/ CD3 complex cross-linking MHC/ peptide, LFA-1, expressed on T cell increases a higher affinity and avidity for ICAM-1 rapidly. LFA-1 is a key molecule in formation of the immune synapse.

T细胞表面LFA1对ICAM1的亲和力及亲合力在TCR/CD3交联MHC/抗原肽后迅速上调,并参与T细胞免疫突触的形成。

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5、
6、

Conclusion JNK/ SAPK may have been aberrantly activated in MSPGN, which may involve in the overexpression of ICAM 1 in MSPGN.

结论MSPGN的肾小球系膜细胞的JNK/SAPK存在异常活化,这种异常活化可能介导了ICAM-1的过度表达。

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Each group was subdivided into 0-hour IR, 3-hour IR and 6-hour IR subgroups. The rats were scarified at corresponding time point under anesthesia. The expression of ICAM-1 was tested by using immunohistochemistry, and the activity of MPO by chromatometry.

各组均又分为再灌注0,3,6h组,再相应时间点麻醉状态下处死大鼠取脑,用免疫组织化学方法检测缺血脑组织细胞间黏附分子1表达,用比色法测定缺血脑组织髓过氧化物酶活性。

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