1、

Conclusion: Through inducing cell cycle retardation in cell cycle and apoptosis, PGG can inhibit proliferation of ovarian carcinoma cells.

结论PGG能抑制卵巢癌细胞的体外增殖,其机制涉及PGG诱导细胞周期S-期阻滞与诱导细胞凋亡。

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Activation of cytokines in the OPG/ RANK/ RANKL signaling pathway, including OPG and RANKL, etc., indirectly inhibit the apoptosis of osteoclasts and promote bone resorption, Subsequently, the spinal vertebrae linear growth was reduced.

通过激活OPG/RANK/RANKL信号通路中多种细胞因子,主要包括OPG和RANKL等,间接抑制破骨细胞的凋亡,进而促进骨吸收,抑制脊柱椎体的线性生长。

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3、

Conclusion: Apoptosis inducement of are some of the mechanisms of BST postponing the CRF process.

结论: 保肾片延缓慢性肾衰进展的作用机理之一与诱导系膜细胞发生凋亡有关.

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4、

The disulfide bond reducing agent dithiothreitol ( DTT) significantly blocked As 2O 3 induced ΔΨ m collapse and apoptosis.

巯基还原剂&二巯基苏糖醇(DTT)显著抑制As2O3诱导的ΔΨm下降。

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5、

After renaturation, AIF protein binded DNA and induced nuclear apoptosis.

经EMSA与 Hoechst33258 染色实验证实:获得的AIF蛋白具有良好地与DNA结合并可诱导肿瘤细胞核凋亡的能力,凋亡率为37%.

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However, wether repairing of damaged telomeres is a mechanism of potentially lethal damage repair has not been clarified until now, and little is understood about the mechanism of telomerase performing repairing at damaged telomeres and modifying apoptosis.

目前对端粒损伤修复是否是潜在致死性损伤修复中重要的机制还未见有学者明确提出,对端粒酶如何参与端粒损伤的修复及凋亡的调控机制了解还很少。

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Through clonal survival, there was radiotherapy enhancement for Glu-GNPs on lung adenocarcinoma cell line A549, and the mechanism may are restrain of repair of sublethal damage, blocking cells at G2/ M and inducing apoptosis.

克隆形成实验表明纳米金对人肺腺癌细胞株A549具有放射增敏作用,初步探讨其机制可能为抑制细胞亚致死损伤的修复阻滞细胞于G2/M期,并诱导细胞凋亡。

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Study on expression of apoptosis and lethal gene during acute rejection in limb allograft

肢体移植排斥反应中细胞凋亡及致死基因表达

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Conclusion HSV-TK/ GCV in combination with HCPT can enhance the lethal effect of suicide gene therapy against human bladder carcinoma cells and effectively induce apoptosis of the cells.

结论HSV-TK/GCV系统联合HCPT治疗能增强自杀基因系统对人膀胱癌的杀伤作用,能够良好地诱导人膀胱癌细胞的凋亡。

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Objective To investigate the effects of resveratrol on inhibiting proliferation and inducing apoptosis of U 251 cells.

目的研究白藜芦醇对人脑胶质瘤细胞U251细胞生长抑制及诱导细胞凋亡的作用.

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11、

Conclusion Ani have a remarkable protection on cerebral ischemia reperfusion damage. Its mechanism may be related with decreasing the neuronal apoptosis.

结论Ani可减少脑缺血再灌注神经元凋亡,提高神经元的存活数目,对脑缺血再灌注损伤起保护作用。

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CREG Modulates Apoptosis of Human Vascular Smooth Muscle Cells via p38/ JNK MAPK Signaling Pathway

CREG通过p38/JNK丝裂原活化蛋白激酶信号通路调控血管平滑肌细胞凋亡

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Following culture of HL 60 with MT, the apoptosis relative cell cycle change appeared in HL 60.This change mainly affected G 2/ M and S stage cells, and presented dose time correlation.

与雷公藤多甙共培养后,HL60发生了与细胞凋亡相关的细胞生物周期变化;此作用主要影响G2/M和S期细胞,并且呈剂量和时间依赖性;

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Experimental research of d-catechin against apoptosis of the liver cells

d-儿茶精抗肝细胞凋亡的实验研究

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Baicalein selectively induce apoptosis in human leukemia K562 cells

黄芩苷元选择性诱导人白血病K562细胞凋亡

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The Effects and Mechanisms of Baicalin on Inducing HL-60 Cells Apoptosis and Growth Inhibition

黄芩苷对HL-60细胞增殖、凋亡、分化的影响及其作用机制的探讨

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Apoptosis of epithelium cells in control was more than in amniotic membrane transplantation.

对照侧上皮细胞的凋亡较羊膜移植侧明显。

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18、

Objective To investigate the apoptosis in neural epithelium induced by passive smoking.

目的探讨被动吸烟对神经管的致畸作用与细胞凋亡的关系。

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As we all known the bcl-2 family protein plays an important role in the apoptosis of neuron death, including the antiapoptotic protein bcl-2 、 bcl-xL 、 Mcl-1 and the proapoptotic protein bax 、 bix etc.

bcl-2家族蛋白在神经元凋亡中起重要的作用,包括抑制凋亡的bcl-2、bcl-xL、Mcl-1和促进凋亡的bax、bix等。

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Reconstruct MCL by semi-tendinous after severe aspiration injury, which could slow down the languishment of cartilage, and decrease apoptosis of chondrocytes.

MCL重度损伤后用半腱肌腱重建MCL能够减慢软骨的退变,减少软骨细胞的凋亡。

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